Original Article from The New England Journal of Medicine — Thrombotic Thrombocytopenia after ChAdOx1 nCov-19 Vaccination
Artigo da nature.
Síndrome pós-aguda COVID-19
Uma revisão abrangente da literatura atual sobre COVID-19 pós-aguda, a sua patofisiologia e as suas sequelas específicas para os órgãos.
SARS-CoV-2, the pathogenic agent of COVID-19, employs angiotensin converting enzyme-2 (ACE2) as its cell entry receptor. Clinical data reveal that in severe COVID-19, SARS-CoV-2 infects the lung, leading to a frequently lethal triad of respiratory insufficiency, acute cardiovascular failure, and coagulopathy.
Elevated D-dimers and lack of anticoagulation predict PE in severe COVID-19 patients | European Respiratory Society
— Ler em erj.ersjournals.com/content/56/4/2001811
This meta-analysis pools data from 7 randomized trials to estimate the association between administration of corticosteroids vs usual care or placebo and all-cause mortality at 28 days in patients with severe coronavirus disease 2019 (COVID-19).
It seemed a truth universally acknowledged that the human population had no pre-existing immunity to SARS-CoV-2, but is that actually the case? Peter Doshi explores the emerging research on immunological responses.
Abstract. The vascular endothelium provides the crucial interface between the blood compartment and tissues, and displays a series of remarkable properties tha
— Ler em academic.oup.com/eurheartj/article/41/32/3038/5901158
A trombose pulmonar parece ser comum na pneumonia COVID-19 e assume duas formas, embolia pulmonar proximal e / ou trombose distal. Os possíveis mecanismos e implicações clínicas são discutidos.